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It has been found that in the brains of AD patients, proteasome activity is reduced, and this change is negatively correlated with the number of NFTs, suggesting that inhibition of proteasome activity prevents the degradation of phosphorylated Tau proteins, which in turn promotes the formation of NFTs (Harris et al., 2020)

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This allows researchers to study acute, pulsatile GH dynamics rather than the sustained tonic elevation produced by DAC-modified CJC-1295, which can mask the natural diurnal pulse structure essential to many endocrine studies

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